![]() Reabsorption of sodium chloride-lessons from the chloride channels. Life-threatening hypokalaemia and lactate accumulation after autointoxication with Stacker 2, a 'powerful slimming agent'. 9(9):821-34.ĭe Wijkerslooth LR, Koch BC, Malingre MM, et al. Endocrine and Metabolic Manifestations of Invasive Fungal Infections and Systemic Antifungal Treatment. Altered fluid, electrolyte and mineral status in tropical disease, with an emphasis on malaria and leptospirosis. Hypokalemia in outpatients with eating disorders. Greenfeld D, Mickley D, Quinlan DM, Roloff P. Changes in Serum Potassium Mediate Thiazide-Induced Diabetes. Shafi, T, Appel LJ, Miller III, ER, Klag MJ, et al. Primary aldosteronism associated with severe rhabdomyolysis due to profound hypokalemia. Toto A, Takahashi Y, Kishimoto M, Minowada S, Aibe H, Hasuo K, et al. Cardiovascular and cerebrovascular comorbidities of hypokalemic and normokalemic primary aldosteronism: results of the German Conn's Registry. 150:387-395.īorn-Frontsberg E, Reincke M, Rump LC, et al. QTc interval screening in methadone treatment. Krantz MJ, Martin J, Stimmel B, Mehta D, Haigney MC. ![]() Molecular Basis of Hypokalemia-Induced Ventricular Fibrillation. Pezhouman A, Singh N, Song Z, Nivala M, Eskandari A, Cao H, et al. Multifaceted persistent hypokalaemia in a patient with coronavirus disease 2019. 100:449-454.īarkas F, Filippas-Ntekouan S, Liontos A, Kosmidou M, Kalambokis G, Milionis H. Hypokalemia as a sensitive biomarker of disease severity and the requirement for invasive mechanical ventilation requirement in COVID-19 pneumonia: A case series of 306 Mediterranean patients. Moreno-P O, Leon-Ramirez JM, Fuertes-Kenneally L, Perdiguero M, Andres M, Garcia-Navarro M, et al. Assessment of Hypokalemia and Clinical Characteristics in Patients With Coronavirus Disease 2019 in Wenzhou, China. Role of muscle in regulating extracellular. Skeletal muscle regulates extracellular potassium. Narrative review: evolving concepts in potassium homeostasis and hypokalemia. Greenlee M, Wingo CS, McDonough AA, Youn JH, Kone BC. Glomerulosa cell-a unique sensor of extracellular K+ concentration. Addison's disease and the regulation of potassium: the role of insulin and aldosterone. Potassium: From Physiology to Clinical Implications. Zacchia M, Abategiovanni ML, Stratigis S, Capasso G. ![]() Mechanism of thyrotoxic periodic paralysis. The result of this test may indicate a serious health condition which should be given immediate medical attention.Castro D, Sharma S. This test includes blood urea nitrogen, creatinine, glucose, carbon dioxide, chloride, sodium, potassium, and calcium. ConclusionīMP test is necessary for you to determine your glucose levels, electrolyte levels, and kidney function. On the other hand, low levels may be due to persistent vomiting, heavy sweating, or diarrhea. Increased levels of potassium may indicate antihypertensive ACE inhibitor medication and kidney failure. This might also lead to nervous problems, arthritis, memory loss, acid reflux, osteoporosis, fatigue, bacterial infections, and kidney stones. While decreased levels may be due to pneumonia, liver disease, diabetes, or lack of sodium in the diet. High levels of sodium may be due to kidney disease, persistent diarrhea, or dehydration. Low levels of chloride may be due to vomiting, heavy sweating, or kidney diseases. Increased levels of chloride may be an indication of overactive parathyroid glands, diarrhea, or kidney diseases. If the result is less than normal, then you may have metabolic acidosis. If your result is above normal, then you may have metabolic alkalosis. Your bicarbonate level may indicate that you have metabolic problems.
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